RGK protein-mediated impairment of slow depolarization- dependent Ca2+entry into developing myotubes
Author:
Publisher
Informa UK Limited
Subject
Biochemistry,Biophysics
Link
http://www.tandfonline.com/doi/pdf/10.4161/chan.27686
Reference27 articles.
1. Voltage Dependent Charge Movement in Skeletal Muscle: a Possible Step in Excitation–Contraction Coupling
2. Involvement of dihydropyridine receptors in excitation–contraction coupling in skeletal muscle
3. Restoration of excitation—contraction coupling and slow calcium current in dysgenic muscle by dihydropyridine receptor complementary DNA
4. Conformational activation of Ca2+ entry by depolarization of skeletal myotubes
5. Ryanodine Receptor Type 1 (RyR1) Mutations C4958S and C4961S Reveal Excitation-coupled Calcium Entry (ECCE) Is Independent of Sarcoplasmic Reticulum Store Depletion
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