Variable maternal methylation overlapping thenc886/vtRNA2-1locus is locked between hypermethylated repeats and is frequently altered in cancer-Reference-Cited by-同舟云学术

Variable maternal methylation overlapping thenc886/vtRNA2-1locus is locked between hypermethylated repeats and is frequently altered in cancer

Published:2014-03-03 Issue:5 Volume:9 Page:783-790
ISSN:1559-2294
Container-title:Epigenetics
language:en
Short-container-title:Epigenetics

Author:

Romanelli Valeria,Nakabayashi Kazuhiko,Vizoso Miguel,Moran Sebastián,Iglesias-Platas Isabel,Sugahara Naoko,Sugahara Naoko,Simón Carlos,Simón Carlos,Hata Kenichiro,Hata Kenichiro,Esteller Manel,Esteller Manel,Court Franck,Court Franck,Monk David,Monk David

Publisher

Informa UK Limited

Subject

Cancer Research,Molecular Biology

Link

http://www.tandfonline.com/doi/pdf/10.4161/epi.28323

Reference26 articles.

1. Genomic surveys by methylation-sensitive SNP analysis identify sequence-dependent allele-specific DNA methylation

2. DNA methylation contributes to natural human variation

3. Dnmt3L and the Establishment of Maternal Genomic Imprints

4. The SRA protein Np95 mediates epigenetic inheritance by recruiting Dnmt1 to methylated DNA

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