Two mammalian MAGOH genes contribute to exon junction complex composition and nonsense-mediated decay
Author:
Publisher
Informa UK Limited
Subject
Cell Biology,Molecular Biology
Link
http://www.tandfonline.com/doi/pdf/10.4161/rna.25827
Reference27 articles.
1. An extensive network of coupling among gene expression machines
2. Pre-mRNA Processing Reaches Back toTranscription and Ahead to Translation
3. Human spliceosomal protein CWC22 plays a role in coupling splicing to exon junction complex deposition and nonsense-mediated decay
4. Human CWC22 escorts the helicase eIF4AIII to spliceosomes and promotes exon junction complex assembly
5. CWC22 Connects Pre-mRNA Splicing and Exon Junction Complex Assembly
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1. MAGOH promotes gastric cancer progression via hnRNPA1 expression inhibition-mediated RONΔ160/PI3K/AKT signaling pathway activation;Journal of Experimental & Clinical Cancer Research;2024-01-25
2. The paralogues MAGOH and MAGOHB are oncogenic factors in high-grade gliomas and safeguard the splicing of cell division and cell cycle genes;RNA Biology;2023-06-09
3. Potential biomarker proteins for aspiration pneumonia detected by shotgun proteomics using buccal mucosa samples: a cross-sectional case–control study;Clinical Proteomics;2023-03-09
4. Advances in the Study of MAGOH and MAGOHB in Malignant Tumors;Advances in Clinical Medicine;2023
5. The paralogues MAGOH and MAGOHB are oncogenic factors in high-grade gliomas and safeguard the splicing of cell division and cell cycle genes;2022-12-20
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