Engineered Kir6.2 mutations that correct the trafficking defect of KATPchannels caused by specific SUR1 mutations
Author:
Publisher
Informa UK Limited
Subject
Biochemistry,Biophysics
Link
http://www.tandfonline.com/doi/pdf/10.4161/chan.25003
Reference26 articles.
1. Reconstitution of I KATP : An Inward Rectifier Subunit Plus the Sulfonylurea Receptor
2. Molecular Biology of Adenosine Triphosphate-Sensitive Potassium Channels
3. ATP-sensitive potassium channelopathies: focus on insulin secretion
4. KATP channels as molecular sensors of cellular metabolism
5. Cloning of the β Cell High-Affinity Sulfonylurea Receptor: a Regulator of Insulin Secretion
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Pharmacological chaperones of ATP-sensitive potassium channels: Mechanistic insight from cryoEM structures;Molecular and Cellular Endocrinology;2020-02
2. Expression, purification, and electrophysiological characterization of a recombinant, fluorescent Kir6.2 in mammalian cells;Protein Expression and Purification;2018-06
3. Structurally Distinct Ligands Rescue Biogenesis Defects of the KATP Channel Complex via a Converging Mechanism;Journal of Biological Chemistry;2015-03
4. Pharmacological rescue of trafficking-impaired ATP-sensitive potassium channels;Frontiers in Physiology;2013
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