CaV1.2 I-II linker structure and Timothy syndrome
Author:
Publisher
Informa UK Limited
Subject
Biochemistry,Biophysics
Link
http://www.tandfonline.com/doi/pdf/10.4161/chan.22078
Reference9 articles.
1. The Role of a Voltage-Dependent Ca2+ Channel Intracellular Linker: A Structure-Function Analysis
2. CaV1.2 Calcium Channel Dysfunction Causes a Multisystem Disorder Including Arrhythmia and Autism
3. The Role of the GX9GX3G Motif in the Gating of High Voltage-activated Ca2+ Channels
4. The Timothy syndrome mutation differentially affects voltage- and calcium-dependent inactivation of CaV1.2 L-type calcium channels
5. Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations
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1. Autism associated mutations in β2 subunit of voltage-gated calcium channels constitutively activate gene expression;Cell Calcium;2022-12
2. A CACNA1C variant associated with cardiac arrhythmias provides mechanistic insights in the calmodulation of L-type Ca2+ channels;Journal of Biological Chemistry;2022-12
3. Timothy syndrome iPSC modeling;Molecular and Cellular Neuroscience;2020-09
4. Elevated basal transcription can underlie timothy channel association with autism related disorders;Progress in Neurobiology;2020-08
5. Cav1.2 channelopathies causing autism: new hallmarks on Timothy syndrome;Pflügers Archiv - European Journal of Physiology;2020-07
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