p27Kip1is required to maintain proliferative quiescence in the adult cochlea and pituitary
Author:
Publisher
Informa UK Limited
Subject
Cell Biology,Developmental Biology,Molecular Biology
Link
http://www.tandfonline.com/doi/pdf/10.4161/cc.10.8.15301
Reference45 articles.
1. p27(Kip1) links cell proliferation to morphogenesis in the developing organ of Corti
2. Gene disruption of p27Kip1 allows cell proliferation in the postnatal and adult organ of Corti
3. Mice Lacking p27Kip1 Display Increased Body Size, Multiple Organ Hyperplasia, Retinal Dysplasia, and Pituitary Tumors
4. Enhanced Growth of Mice Lacking the Cyclin-Dependent Kinase Inhibitor Function of p27Kip1
5. A Syndrome of Multiorgan Hyperplasia with Features of Gigantism, Tumorigenesis, and Female Sterility in p27Kip1-Deficient Mice
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3. Expression of Atoh1 , Gfi1 , and Pou4f3 in the mature cochlea reprograms nonsensory cells into hair cells;Proceedings of the National Academy of Sciences;2024-01-24
4. SoxC transcription factors shape the epigenetic landscape to establish competence for sensory differentiation in the mammalian organ of Corti;Proceedings of the National Academy of Sciences;2023-08-16
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