The TIF1α-related TRIM cofactors couple chromatin modifications to transcriptional regulation, signaling and tumor suppression
Author:
Publisher
Informa UK Limited
Subject
Genetics,Biochemistry,Biotechnology
Link
http://www.tandfonline.com/doi/pdf/10.4161/trns.2.5.17725
Reference32 articles.
1. The N-terminal part of TIF1, a putative mediator of the ligand-dependent activation function (AF-2) of nuclear receptors, is fused to B-raf in the oncogenic protein T18.
2. TRIM24 links a non-canonical histone signature to breast cancer
3. Recruitment of TIF1γ to Chromatin via Its PHD Finger-Bromodomain Activates Its Ubiquitin Ligase and Transcriptional Repressor Activities
4. PHD Domain-Mediated E3 Ligase Activity Directs Intramolecular Sumoylation of an Adjacent Bromodomain Required for Gene Silencing
5. Structural insights into human KAP1 PHD finger–bromodomain and its role in gene silencing
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