Genetic disruption ofUSP9Xsensitizes colorectal cancer cells to 5-fluorouracil
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Pharmacology,Oncology,Molecular Medicine
Link
http://www.tandfonline.com/doi/pdf/10.4161/cbt.21792
Reference22 articles.
1. Diagnosing and exploiting cancer's addiction to blocks in apoptosis
2. Deubiquitinase USP9X stabilizes MCL1 and promotes tumour cell survival
3. Zymophagy, a Novel Selective Autophagy Pathway Mediated by VMP1-USP9x-p62, Prevents Pancreatic Cell Death*
4. The Ubiquitin Ligase Itch Is Auto-ubiquitylated in Vivo and in Vitro but Is Protected from Degradation by Interacting with the Deubiquitylating Enzyme FAM/USP9X
5. The HECT Family of E3 Ubiquitin Ligases: Multiple Players in Cancer Development
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2. Cellular functions, molecular signalings and therapeutic applications: Translational potential of deubiquitylating enzyme USP9X as a drug target in cancer treatment;Biochimica et Biophysica Acta (BBA) - Reviews on Cancer;2024-05
3. Comprehensive analysis of the role of ubiquitin-specific peptidases in colorectal cancer: A systematic review;World Journal of Gastrointestinal Oncology;2024-01-15
4. Ribosome Quality Control Mechanism Mitigates the Cytotoxic Impacts of Ribosome Collisions Induced by 5-Fluorouracil;2023-12-26
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