Resistance to 9-3-D-Arabinofuranosyl-2-Fluoroadenine due to Reduced Incorporation into DNA from Competition by Excess Deoxyadenosine Triphosphate: Implications for Different Sensitivities to Nucleoside Analogues-Reference-Cited by-同舟云学术

Resistance to 9-3-D-Arabinofuranosyl-2-Fluoroadenine due to Reduced Incorporation into DNA from Competition by Excess Deoxyadenosine Triphosphate: Implications for Different Sensitivities to Nucleoside Analogues

Published:2005-06-01 Issue:5 Volume:81 Page:405-412
ISSN:0925-5710
Container-title:International Journal of Hematology
language:
Short-container-title:International Journal of Hematology

Author:

Yoshio Nobuyuki,Kawai Yasukazu,Hori Hiroki,Ueda Takanori

Publisher

Springer Science and Business Media LLC

Subject

Hematology

Link

http://www.springerlink.com/index/pdf/10.1532/IJH97.05008

Reference27 articles.

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3. Takagi K, Kawai Y, Yamauchi T, Ueda T. Inhibition of repair of carboplatin-induced DNA damage by 9-3-D-arabinofuranosyl-2-fluoroadenine in quiescent human lymphocytes. Biochem Pharmacol. 2004;68:1757-1766.

4. Yamauchi T, Nowak BJ, Keating MJ, Plunkett W. DNA repair initiated in chronic lymphocytic leukemia lymphocytes by 4-hydroperoxycyclophosphamide is inhibited by fludarabine and clofarabine. Clin Cancer Res. 2001;7:3580-3589.

5. Plunkett W, Huang P, Gandhi V. Metabolism and action of fludarabine phosphate. Semin Oncol. 1990;17:3-17.

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