A Novel Mutation in the Juxtamembrane Intracellular Sequence of the Granulocyte Colony-Stimulating Factor (G-CSF) Receptor Gene in a Patient with Severe Congenital Neutropenia Augments G-CSF Proliferation Activity but Not through the MAP Kinase Cascade-Reference-Cited by-同舟云学术

A Novel Mutation in the Juxtamembrane Intracellular Sequence of the Granulocyte Colony-Stimulating Factor (G-CSF) Receptor Gene in a Patient with Severe Congenital Neutropenia Augments G-CSF Proliferation Activity but Not through the MAP Kinase Cascade

Published:2005-07-01 Issue:1 Volume:82 Page:28-34
ISSN:0925-5710
Container-title:International Journal of Hematology
language:
Short-container-title:International Journal of Hematology

Author:

Yokoyama Toshihiro,Okamura Seiichi,Asano Yoshinobu,Kamezaki Kenjirou,Numata Akihiko,Kakumitsu Haruko,Shide Koutarou,Nakashima Hitoshi,Kanaji Taisuke,Sekine Yuichi,Mizuno Yumi,Okamura Jun,Matsuda Tadashi,Harada Mine,Niho Yoshiyuki,Shimoda Kazuya

Publisher

Springer Science and Business Media LLC

Subject

Hematology

Link

http://www.springerlink.com/index/pdf/10.1532/IJH97.05010

Reference38 articles.

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3. Lee CK, Raz R, Gimeno R, et al. STAT3 is a negative regulator of granulopoiesis but is not required for G-CSF-dependent differentiation. Immunity. 2002;17:63-72.

4. Kamezaki K, Shimoda K, Numata A, et al. Roles of Stat3 and ERK in G-CSF signaling. Stem Cells. 2005;23:252-263.

5. Meraz MA, White JM, Sheehan KC, et al. Targeted disruption of the Stat1 gene in mice reveals unexpected physiologic specificity in the JAK-STAT signaling pathway. Cell. 1996;84:431-442.

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