Case of an 18-year-old Female with EML4-ALK-positive Lung Adenocarcinoma
Author:
Publisher
Japan Lung Cancer Society
Subject
Pulmonary and Respiratory Medicine,Oncology
Link
https://www.jstage.jst.go.jp/article/haigan/54/3/54_153/_pdf
Reference15 articles.
1. 1. Soda M, Choi YL, Enomoto M, Takada S, Yamashita Y, Ishikawa S, et al. Identification of the transforming EML4-ALK fusion gene in non-small-cell lung cancer. Nature. 2007; 448: 561-566.
2. 2. Shaw AT, Kim DW, Nakagawa K, Seto T, Crinó L, Ahn MJ, et al. Crizotinib versus chemotherapy in advanced ALK-positive lung cancer. N Engl J Med. 2013; 368: 2385-2394.
3. 3. Kim SJ, Kim DW, Kim TM, Lee SH, Heo DS, Bang YJ. Remarkable tumor response to crizotinib in a 14-year-old girl with ALK-positive non-small-cell lung cancer. J Clin Oncol. 2012; 30: e147-e150.
4. 4. Koivunen JP, Mermel C, Zejnullahu K, Murphy C, Lifshits E, Holmes AJ, et al. EML4-ALK fusion gene and efficacy of an ALK kinase inhibitor in lung cancer. Clin Cancer Res. 2008; 14: 4275-4283.
5. 5. Inamura K, Takeuchi K, Togashi Y, Hatano S, Ninomiya H, Motoi N, et al. EML4-ALK lung cancers are characterized by rare other mutations, a TTF-1 cell lineage, an acinar histology, and young onset. Mod Pathol. 2009; 22: 508-515.
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1. A Surgical Case of a 15-year-old Girl with EML4-ALK-positive Lung Adenocarcinoma Detected with Roentgenographic Screening;Haigan;2020-08-20
2. A Case of Lung Cancer in a Teenager;Haigan;2019-10-20
3. Corticosteroid Therapy for Crizotinib-induced, Allergy-mediated Liver Injury: a Case Report;Haigan;2015
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