New findings and new methods on macrophages in primary immune thrombocytopenia-Reference-Cited by-同舟云学术

New findings and new methods on macrophages in primary immune thrombocytopenia

Published:2024-05-03 Issue:6 Volume:204 Page:2157-2158
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:Br J Haematol

Author:

Ji Lili¹,Zhan Yanxia¹,Cheng Yunfeng¹²³⁴^ORCID

Affiliation:

1. Department of Hematology Zhongshan Hospital, Fudan University Shanghai China

2. Institute of Clinical Science, Zhongshan Hospital, Fudan University Shanghai China

3. Department of Hematology Zhongshan Hospital Qingpu Branch, Fudan University Shanghai China

4. Center for Tumor Diagnosis & Therapy Jinshan Hospital, Fudan University Shanghai China

Abstract

Primary immune thrombocytopenia (ITP) is an acquired autoimmune disease with multiple immune cells take part in the pathogenesis. Macrophages play multiple roles in both innate and adaptive immune system. The report by Jiani Mo and colleagues identified new biomarkers and explore the role of mitophagy and ferroptosis in ITP pathogenesis.Commentary on: Mo et al. Comprehensive analysis and prediction model of mitophagy and ferroptosis in primary immune thrombocytopenia. Br J Haematol 2024;204:2429‐2442.

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjh.19513

Reference9 articles.

1. Serum from half of patients with immune thrombocytopenia triggers macrophage phagocytosis of platelets

2. MST4 kinase regulates immune thrombocytopenia by phosphorylating STAT1-mediated M1 polarization of macrophages

3. Low‐dose decitabine promotes M2 macrophage polarization in patients with primary immune thrombocytopenia via enhancing KLF4 binding to PPARγ promoter

4. Protective and pathogenic functions of macrophage subsets

5. IRF5 promotes inflammatory macrophage polarization and TH1-TH17 responses

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