Smad3 gene C‐terminal phosphorylation site mutation aggravates CCl 4 ‐induced inflammation in mice
Author:
Affiliation:
1. Department of Pharmacology Key Laboratory of Anti‐inflammatory and Immunopharmacology Ministry of Education Anhui Medical University Hefei China
2. Department of Anatomy Anhui Medical University Hefei China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.15385
Reference42 articles.
1. Targeting TGF-β Mediated SMAD Signaling for the Prevention of Fibrosis
2. Central role of dysregulation of TGF-β/Smad in CKD progression and potential targets of its treatment
3. Compound Astragalus and Salvia miltiorrhiza extracts modulate MAPK-regulated TGF-β/Smad signaling in hepatocellular carcinoma by multi-target mechanism
4. Pathobiology of liver fibrosis: a translational success story
5. New insights into TGF-β/Smad signaling in tissue fibrosis
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. The Role of TGF-β/SMAD Signaling in Hepatocellular Carcinoma: from Mechanism to Therapy and Prognosis;International Journal of Biological Sciences;2024
2. Amelioratory effects of astragaloside IV on hepatocarcinogenesis via Nrf2-mediated pSmad3C/3L transformation;Phytomedicine;2023-08
3. Smad3 gene C-terminal phosphorylation site mutation exacerbates CCl4-induced hepatic fibrogenesis by promoting pSmad2L/C-mediated signaling transduction;Naunyn-Schmiedeberg's Archives of Pharmacology;2021-06-30
4. Smad3 C-terminal phosphorylation site mutation attenuates the hepatoprotective effect of salvianolic acid B against hepatocarcinogenesis;Food and Chemical Toxicology;2021-01
5. Polydeoxyribonucleotide Exerts Protective Effect Against CCl4-Induced Acute Liver Injury Through Inactivation of NF-κB/MAPK Signaling Pathway in Mice;International Journal of Molecular Sciences;2020-10-24
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3