Rae1 drives NKG2D binding‐dependent tumor development in mice by activating mTOR and STAT3 pathways in tumor cells-Reference-Cited by-同舟云学术

Rae1 drives NKG2D binding‐dependent tumor development in mice by activating mTOR and STAT3 pathways in tumor cells

Published:2020-05-16 Issue:7 Volume:111 Page:2234-2247
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Sci

Author:

Zhao Peiyan¹,Yang Lei¹,Li Xin¹,Lu Wenting¹,Lu Fangjie¹,Wang Shengnan¹,Wang Ying¹,Hua Li²,Cui Cuiyun²,Dong Boqi²,Yu Yongli²,Wang Liying¹^ORCID

Affiliation:

1. Department of Molecular Biology College of Basic Medical Sciences and Institute of Pediatrics The First Hospital of Jilin University Jilin University Changchun China

2. Department of Immunology College of Basic Medical Sciences Jilin University Changchun China

Funder

Natural Science Foundation of Jilin Province

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cas.14434

Reference51 articles.

1. Roles of the NKG2D immunoreceptor and its ligands

2. Regulation of Ligands for the NKG2D Activating Receptor

3. NKG2D-Deficient Mice Are Defective in Tumor Surveillance in Models of Spontaneous Malignancy

4. NKG2D–NKG2D Ligand Interaction Inhibits the Outgrowth of Naturally Arising Low-Grade B Cell Lymphoma In Vivo

5. Activated and expanded natural killer cells target osteosarcoma tumor initiating cells in an NKG2D–NKG2DL dependent manner

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