Intense orthodontic force induces the three dental pulp nitric oxide synthase isoforms and leads to orofacial discomfort in rats-Reference-Cited by-同舟云学术

Intense orthodontic force induces the three dental pulp nitric oxide synthase isoforms and leads to orofacial discomfort in rats

Published:2022-01-14 Issue: Volume: Page:
ISSN:1601-6335
Container-title:Orthodontics & Craniofacial Research
language:en
Short-container-title:Orthod Craniofacial Res

Author:

Sobrinho José Edson Moreira¹,Aguiar Maria Thaynara¹,Machado Larissa Carvalho¹,Carlos Anna Clara Aragão Matos¹,Nunes Alves Ana Paula Negreiros²,Mesquita Karine Cestaro¹,Miranda Candeiro George Táccio¹,Barros Silva Paulo Goberlânio¹^ORCID

Affiliation:

1. Department of Dentistry Laboratory of Pathology Christus University Center Fortaleza Brazil

2. Laboratory of Oral Pathology Federal University of Ceará Fortaleza Brazil

Publisher

Wiley

Subject

Otorhinolaryngology,Oral Surgery,Surgery,Orthodontics

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/ocr.12560

Reference35 articles.

1. The role of osteocytes during experimental orthodontic tooth movement: A review

2. Pulpal Reactions to Orthodontic Force Application in Humans: A Systematic Review

3. Interleukin-17 is involved in orthodontically induced inflammatory root resorption in dental pulp cells

4. Orthodontic treatment mediates dental pulp microenvironment via IL17A

5. Exogenous nitric oxide stimulates the odontogenic differentiation of rat dental pulp stem cells

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1. Role of induced nitric oxide synthases in orofacial nociception/discomfort after dental tooth bleaching with hydrogen peroxide;Archives of Oral Biology;2024-05

2. Endodontic–orthodontic interactions: a review and treatment recommendations;Australian Dental Journal;2023-06

3. Interleukin-17 plays a role in dental pulp inflammation mediated by zoledronic acid: a mechanism unrelated to the Th17 immune response?;Journal of Applied Oral Science;2023

4. The Inhibition of the Inducible Nitric Oxide Synthase Enhances the DPSC Mineralization under LPS-Induced Inflammation;International Journal of Molecular Sciences;2022-11-23