Urokinase‐type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)‐mediated sodium retention in experimental nephrotic syndrome-Reference-Cited by-同舟云学术

Urokinase‐type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)‐mediated sodium retention in experimental nephrotic syndrome

Published:2019-05-20 Issue:4 Volume:227 Page:
ISSN:1748-1708
Container-title:Acta Physiologica
language:en
Short-container-title:Acta Physiol

Author:

Bohnert Bernhard N.¹²³,Daiminger Sophie¹,Wörn Matthias¹,Sure Florian⁴,Staudner Tobias⁴,Ilyaskin Alexandr V.⁴,Batbouta Firas¹,Janessa Andrea¹,Schneider Jonas C.¹,Essigke Daniel¹,Kanse Sandip⁵,Haerteis Silke⁶,Korbmacher Christoph⁴,Artunc Ferruh¹²³^ORCID

Affiliation:

1. Department of Internal Medicine, Division of Endocrinology, Diabetology, Vascular Disease, Nephrology and Clinical Chemistry University Hospital Tübingen Tübingen Germany

2. Institute of Diabetes Research and Metabolic Diseases (IDM) of the Helmholtz Center Munich at the University Tübingen Tübingen Germany

3. German Center for Diabetes Research (DZD) at the University Tübingen Tübingen Germany

4. Institute of Cellular and Molecular Physiology Friedrich‐Alexander University Erlangen‐Nürnberg (FAU) Bayern Germany

5. Institute of Basic Medical Sciences University of Oslo Oslo Norway

6. Institute of Anatomy University of Regensburg Regensburg Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Physiology

Link