Interleukin 8 and granulocyte elastase α1 proteinase inhibitor complex in the tracheobronchial aspirate of infants with chronic lung disease following respiratory distress syndrome
Author:
Publisher
Wiley
Subject
Pediatrics, Perinatology, and Child Health
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1442-200X.1996.tb03522.x/fullpdf
Reference22 articles.
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2. Neutrophil-activating peptide-1/interleukin 8, a novel cytokine that activates neutrophils;Baggiolini;J. Clin. Invest.,1989
3. Purification and partial biochemical characterization of a human monocyte-derived neutrophil-activating peptide that lacks interleukin 1 activity;Schroeder;J. Immunol.,1987
4. Stable microbubble test for predicting the risk of respiratory distress syndrome: I. Comparisons with other predictors of fetal lung maturity in amniotic fluid;Chida;Eur. J. Pediatr.,1993
5. Incidence and evolution of subependymal and intraventricular hemorrhage: A study of infants with birth weights less than 1500 g;Papile;J. Pediatr.,1978
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1. Inflammatory changes in the lungs of premature infants with symptomatic patent ductus arteriosus;Pediatrics International;2002-08
2. Anti-interleukin-8 autoantibody in the tracheobronchial aspirate of infants with chronic lung disease;Pediatrics International;2001-02-28
3. Interleukin 8 in the tracheobronchial aspirate of infants acts as a neutrophil chemotactic factor in the development of chronic lung disease;Pediatrics International;1999-02
4. Strategy for the prevention and treatment of chronic lung disease of the premature infant;Pediatric Pulmonology;1999
5. Interleukin 8 and granulocyte elastase in the tracheobronchial aspirate of infants without respiratory distress syndrome or intrauterine infection and development of chronic lung disease;Pediatrics International;1997-08
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