Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws-Reference-Cited by-同舟云学术

Extension of the mitochondria dysfunction hypothesis of metabolic syndrome to atherosclerosis with emphasis on the endocrine-disrupting chemicals and biophysical laws

Published:2013-01 Issue:1 Volume:4 Page:19-33
ISSN:2040-1116
Container-title:Journal of Diabetes Investigation
language:en
Short-container-title:J Diabetes Invest

Author:

Lee Hong Kyu¹,Shim Eun Bo²

Affiliation:

1. Department of Internal Medicine; Eulji University College of Medicine; Seoul Korea

2. Department of Mechanical and Biomedical Engineering; Kangwon National University; Chuncheon Korea

Funder

RIC

Publisher

Wiley

Subject

General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jdi.12048/fullpdf

Reference102 articles.

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4. A strong dose-response relation between serum concentrations of persistent organic pollutants and diabetes: results from the National Health and Examination Survey 1999-2002;Lee;Diabetes Care,2006

5. Chronic exposure to the herbicide, atrazine, causes mitochondrial dysfunction and insulin resistance;Lim;PLoS ONE,2009

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