Enhancedin vitroCA1 network activity in a sodium channel β1(C121W) subunit model of genetic epilepsy
Author:
Affiliation:
1. The Florey Institute of Neuroscience and Mental Health; University of Melbourne; Parkville Victoria Australia
2. Centre for Neural Engineering; University of Melbourne; Parkville Victoria Australia
Funder
National Health and Medical Research Council
Publisher
Wiley
Subject
Clinical Neurology,Neurology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/epi.12568/fullpdf
Reference39 articles.
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2. Na(v)1.1 localizes to axons of parvalbumin-positive inhibitory interneurons: a circuit basis for epileptic seizures in mice carrying an Scn1a gene mutation;Ogiwara;J Neurosci,2007
3. Reduced cortical inhibition in a mouse model of familial childhood absence epilepsy;Tan;Proc Natl Acad Sci USA,2007
4. Axon initial segment dysfunction in a mouse model of genetic epilepsy with febrile seizures plus;Wimmer;J Clin Invest,2010
5. Reduced sodium current in GABAergic interneurons in a mouse model of severe myoclonic epilepsy in infancy;Yu;Nat Neurosci,2006
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