Could the PI3K canonical pathway be a common link between chronic inflammatory conditions and oral carcinogenesis?-Reference-Cited by-同舟云学术

Could the PI3K canonical pathway be a common link between chronic inflammatory conditions and oral carcinogenesis?

Published:2016-03-15 Issue:7 Volume:45 Page:469-474
ISSN:0904-2512
Container-title:Journal of Oral Pathology & Medicine
language:en
Short-container-title:J Oral Pathol Med

Author:

Sonis Stephen T.¹,Amaral Mendes Rui²^ORCID

Affiliation:

1. Brigham and Women's Hospital and the Dana-Farber Cancer Institute and Biomodels LLC; Boston MA USA

2. Adjunct Professor of Oral and Maxillofacial Medicine; Department of Oral and Maxillofacial Medicine and Diagnostic Sciences; School of Dental Medicine; Case Western Reserve University; Cleveland OH USA

Publisher

Wiley

Subject

Periodontics,Cancer Research,Otorhinolaryngology,Oral Surgery,Pathology and Forensic Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jop.12436/fullpdf

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4. Effects of activated fibroblasts on phenotype modulation, EGFR signalling and cell cycle regulation in OSCC cells;Berndt;Exp Cell Res,2014

5. RAS/ PI3K crosstalk and cetuximab resistance in head and neck squamous cell carcinoma;Rampias;Clin Cancer Res,2014

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