Salvage therapy using FLT3 inhibitors may improve long-term outcome of relapsed or refractory AML in patients withFLT3-ITD
Author:
Affiliation:
1. Department of Leukemia; MD Anderson Cancer Center; The University of Texas; Houston; TX; USA
2. Department of Hematopathology; MD Anderson Cancer Center; The University of Texas; Houston; TX; USA
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/bjh.12299/fullpdf
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3. Phase IIB trial of oral Midostaurin (PKC412), the FMS-like tyrosine kinase 3 receptor (FLT3) and multi-targeted kinase inhibitor, in patients with acute myeloid leukemia and high-risk myelodysplastic syndrome with either wild-type or mutated FLT3;Fischer;Journal of Clinical Oncology,2010
4. Prognostic significance of activating FLT3 mutations in younger adults (16 to 60 years) with acute myeloid leukemia and normal cytogenetics: a study of the AML Study Group Ulm;Frohling;Blood,2002
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