High‐throughput analysis revealed the unique immunoglobulin gene rearrangements in plasmacytoma‐like post‐transplant lymphoproliferative disorder-Reference-Cited by-同舟云学术

High‐throughput analysis revealed the unique immunoglobulin gene rearrangements in plasmacytoma‐like post‐transplant lymphoproliferative disorder

Published:2020-03-19 Issue:4 Volume:189 Page:
ISSN:0007-1048
Container-title:British Journal of Haematology
language:en
Short-container-title:Br J Haematol

Author:

Hoshino Akihiro¹^ORCID,Nishimura Akira¹,Naruto Takuya¹,Okano Tsubasa¹,Matsumoto Kazuaki¹,Okamoto Keisuke¹,Shintaku Hiroshi²,Tokoro Shown³,Okamoto Hiroyuki⁴,Wada Taizo⁴,Takagi Masatoshi¹^ORCID,Imai Kohsuke⁵,Kanegane Hirokazu⁶,Morio Tomohiro¹

Affiliation:

1. Department of Pediatrics and Developmental Biology Graduate School of Medical and Dental Sciences Tokyo Medical and Dental University Tokyo Japan

2. Division of Pathology Tokyo Medical and Dental University Hospital Tokyo Japan

3. Department of Dermatology Graduate School of Medicine and Dental Sciences Tokyo Medical and Dental University Tokyo Japan

4. Department of Pediatrics School of Medicine Institute of Medical, Pharmaceutical and Health Sciences Kanazawa University Kanazawa Japan

5. Department of Community Pediatrics, Perinatal and Maternal Medicine Graduate School of Medical and Dental Sciences Tokyo Medical and Dental University Tokyo Japan

6. Department of Child Health and Development Graduate School of Medical and Dental Sciences Tokyo Medical and Dental University Tokyo Japan

Funder

JSPS KAKENHI

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjh.16583

Reference16 articles.

1. Post-transplant lymphoproliferative disorders: From epidemiology to pathogenesis-driven treatment

2. Survival Analyses and Prognosis of Plasma-Cell Myeloma and Plasmacytoma-Like Posttransplantation Lymphoproliferative Disorders

3. Hematopoietic Stem Cell Transplantation for XIAP Deficiency in Japan

4. Wnt3/RhoA/ROCK signaling pathway is involved in adhesion-mediated drug resistance of multiple myeloma in an autocrine mechanism

5. Identification of novel mutational drivers reveals oncogene dependencies in multiple myeloma

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