Delayed ischaemic preconditioning in the presence of galectin-9 protects against renal ischaemic injury through a regulatory T-cell dependent mechanism-Reference-Cited by-同舟云学术

Delayed ischaemic preconditioning in the presence of galectin-9 protects against renal ischaemic injury through a regulatory T-cell dependent mechanism

Published:2016-09-12 Issue:10 Volume:21 Page:828-834
ISSN:1320-5358
Container-title:Nephrology
language:en
Short-container-title:Nephrology

Author:

Zhang Bing-Ying¹,Fang Yi¹²³,Jiao Xiao-Yan¹,Wu Sheng¹,Cai Jie-Ru¹,Zou Jian-Zhou¹²,Ding Xiao-Qiang¹²³

Affiliation:

1. Department of Nephrology, Zhongshan Hospital; Fudan University; Shanghai China

2. Shanghai Institute of Kidney and Dialysis; Shanghai China

3. Shanghai Key Laboratory of Kidney and Blood Purification; Shanghai China

Funder

National Natural Science Foundation of China

Science and Technology Commission of Shanghai Municipality

Publisher

Wiley

Subject

Nephrology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/nep.12680/fullpdf

Reference24 articles.

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4. Regulatory T cells suppress innate immunity in kidney ischemia-reperfusion injury;Kinsey;J. Am. Soc. Nephrol.,2009

5. Regulatory T cells contribute to the protective effect of ischemic preconditioning in the kidney;Kinsey;Kidney Int.,2010

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1. Gal-9/Tim-3 signaling pathway activation suppresses the generation of Th17 cells and promotes the induction of Foxp3+ regulatory T cells in renal ischemia-reperfusion injury;Molecular Immunology;2023-04

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3. Galectins as potential pharmacological targets in renal injuries of diverse etiology;European Journal of Pharmacology;2020-08

4. Superagonistic CD28 protects against renal ischemia injury induced fibrosis through a regulatory T-cell expansion dependent mechanism;BMC Nephrology;2019-11-09

5. The Sweet-Side of Leukocytes: Galectins as Master Regulators of Neutrophil Function;Frontiers in Immunology;2019-08-07