Interleukin 17C is elevated in lesional tissue of hidradenitis suppurativa
Author:
Affiliation:
1. Laboratory of Investigative Dermatology The Rockefeller University New York NY U.S.A.
2. Weill Cornell/Rockefeller/Sloan Kettering Tri‐Institutional MD‐PhD Program New York NY U.S.A.
Funder
National Center for Advancing Translational Sciences
National Institute of General Medical Sciences
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.18556
Reference7 articles.
1. IL-17C regulates the innate immune function of epithelial cells in an autocrine manner
2. IL-17C: A Unique Epithelial Cytokine with Potential for Targeting across the Spectrum of Atopic Dermatitis and Psoriasis
3. Psoriasis pathogenesis and the development of novel targeted immune therapies
4. The IL-1 Pathway Is Hyperactive in Hidradenitis Suppurativa and Contributes to Skin Infiltration and Destruction
5. Apocrine Gland–Rich Skin Has a Non-Inflammatory IL-17–Related Immune Milieu, that Turns to Inflammatory IL-17–Mediated Disease in Hidradenitis Suppurativa
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1. An Atlas of the Hidradenitis Suppurativa Transcriptome;Dermatology and Therapy;2024-01-06
2. Biologics for Hidradenitis suppurativa: evolution of the treatment paradigm;Expert Review of Clinical Immunology;2023-12-22
3. IL-17 Inhibition: A Valid Therapeutic Strategy in the Management of Hidradenitis Suppurativa;Pharmaceutics;2023-10-11
4. Hidradenitis suppurativa: new insights into disease mechanisms and an evolving treatment landscape;British Journal of Dermatology;2023-09-16
5. Single-cell transcriptomics suggest distinct upstream drivers of IL-17A/F in hidradenitis versus psoriasis;Journal of Allergy and Clinical Immunology;2023-09
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