Modeling pathogenesis and treatment response in childhood absence epilepsy-Reference-Cited by-同舟云学术

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Modeling pathogenesis and treatment response in childhood absence epilepsy

Published:2017-12-18 Issue:1 Volume:59 Page:135-145
ISSN:0013-9580
Container-title:Epilepsia
language:en
Short-container-title:Epilepsia

Author:

Knox Andrew T.¹^ORCID,Glauser Tracy²³,Tenney Jeffrey²³,Lytton William W.⁴⁵,Holland Katherine²³

Affiliation:

1. Department of Neurology; University of Wisconsin; Madison WI USA

2. Comprehensive Epilepsy Center; Division of Neurology; Cincinnati Children's Hospital Medical Center; Cincinnati OH USA

3. The University of Cincinnati College of Medicine; Cincinnati OH USA

4. Departments of Neurology and Physiology & Pharmacology; SUNY Downstate Medical Center; Brooklyn NY USA

5. Department Neurology; Kings County Hospital Center; Brooklyn NY USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/epi.13962/fullpdf

Reference37 articles.

1. The current state of absence epilepsy: can we have your attention?;Tenney;Epilepsy Curr,2013

2. Ethosuximide, valproic acid, and lamotrigine in childhood absence epilepsy;Glauser;N Engl J Med,2010

3. The heredity of epilepsy as told by relatives and twins;Lennox;J Am Med Assoc,1951

4. Genes and molecular mechanisms involved in the epileptogenesis of idiopathic absence epilepsies;Yalcin;Seizure,2012

5. Selective increase in T-type calcium conductance of reticular thalamic neurons in a rat model of absence epilepsy;Tsakiridou;J Neurosci,1995

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