Threonine532 phosphorylation in ClC-3 channels is required for angiotensin II-induced Cl−current and migration in cultured vascular smooth muscle cells
Author:
Affiliation:
1. Department of Pharmacology, Cardiac and Cerebral Vascular Research Center, Zhongshan School of Medicine; Sun Yat-Sen University; Guangzhou China
Funder
National Natural Science Foundation of China
CHINA-CANADA Joint Health Research Program
Publisher
Wiley
Subject
Pharmacology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13385/fullpdf
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3. Secretion and cell volume regulation by salivary acinar cells from mice lacking expression of the Clcn3 Cl− channel gene;Arreola;J Physiol,2002
4. Sequence and structure-based prediction of eukaryotic protein phosphorylation sites;Blom;J Mol Biol,1999
5. Angiotensin II (AT1) receptors and NADPH oxidase regulate Cl− current elicited by beta1 integrin stretch in rabbit ventricular myocytes;Browe;J Gen Physiol,2004
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