Deficiency in the DNA repair protein ERCC1 triggers a link between senescence and apoptosis in human fibroblasts and mouse skin-Reference-Cited by-同舟云学术

Deficiency in the DNA repair protein ERCC1 triggers a link between senescence and apoptosis in human fibroblasts and mouse skin

Published:2019-11-18 Issue:3 Volume:19 Page:
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Kim Dong Eun¹^ORCID,Dollé Martijn E. T.²,Vermeij Wilbert P.³⁴,Gyenis Akos⁵,Vogel Katharina⁵,Hoeijmakers Jan H. J.³⁴⁵,Wiley Christopher D.¹,Davalos Albert R.¹,Hasty Paul⁶^ORCID,Desprez Pierre‐Yves¹,Campisi Judith¹⁷

Affiliation:

1. Buck Institute for Research on Aging Novato CA USA

2. Centre for Health Protection Research National Institute of Public Health and the Environment (RIVM) Bilthoven The Netherlands

3. Department of Molecular Genetics Erasmus University Medical Center Rotterdam The Netherlands

4. Princess Máxima Center for Pediatric Oncology ONCODE Institute Utrecht The Netherlands

5. CECAD Forschungszentrum Köln Germany

6. Department of Molecular Medicine Sam and Ann Barshop Institute for Longevity and Aging Studies University of Texas Health Science Center San Antonio TX USA

7. Lawrence Berkeley National Laboratory Berkeley CA USA

Funder

National Institute on Aging

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.13072

Reference44 articles.

1. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders