Physalin B attenuates liver fibrosis via suppressing LAP2α‐HDAC1 mediated deacetylation of glioma‐associated oncogene 1 and hepatic stellate cell activation
Author:
Affiliation:
1. Institute of Molecular Pathobiochemistry, Experimental Gene Therapy and Clinical Chemistry (IFMPEGKC) RWTH University Hospital Aachen Aachen Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Wiley
Subject
Pharmacology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bph.15588
Reference5 articles.
1. Hepatoprotective and Anti-fibrotic Agents: It's Time to Take the Next Step
2. Recent advances in understanding liver fibrosis: bridging basic science and individualized treatment concepts
3. Physalin D attenuates hepatic stellate cell activation and liver fibrosis by blocking TGF-β/Smad and YAP signaling
4. Physalin B ameliorates nonalcoholic steatohepatitis by stimulating autophagy and NRF2 activation mediated improvement in oxidative stress
5. Physalin B attenuates liver fibrosis via suppressing LAP2α–HDAC1‐mediated deacetylation of the transcription factor GLI1 and hepatic stellate cell activation
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1. TWEAK promotes inflammatory response in liver fibrosis;Journal of Biochemical and Molecular Toxicology;2023-07-28
2. Inhibition of long noncoding RNA Gm41724 alleviates pressure overload-induced cardiac fibrosis by regulating lamina-associated polypeptide 2α;Pharmacological Research;2023-02
3. Comprehensive analyses reveal the role of histone deacetylase genes in prognosis and immune response in low-grade glioma;PLOS ONE;2022-10-13
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