The CC16 A38G polymorphism is associated with the development of asthma in children with allergic rhinitis
Author:
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2222.2010.03679.x/fullpdf
Reference27 articles.
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3. Gene-environment interactions in asthma and allergie;Martinez;a new paradigm to understand disease causation,2005
4. ACE gene polymorphism might disclose why some Taiwanese children with allergic rhinitis develop asthma symptoms but others do not;Lue;Pediatr Allerg Immunol,2006
5. Demonstration of different forms of the anti-inflammatory proteins lipocortin-1 and Clara cell protein-16 in human nasal and bronchoalveolar lavage fluids;Lindahl;Electrophoresis,1999
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1. Effects of lifestyle and associated diseases on serum CC16 suggest complex interactions among metabolism, heart and lungs;Journal of Advanced Research;2023-06
2. Multimorbidities in Allergic Rhinitis—Current Evidence from Epidemiological Studies, Treatment Trials, and Molecular Data;Current Allergy and Asthma Reports;2023-01-24
3. Noninvasive integrative approach applied to children in the context of recent air pollution exposure demonstrates association between fractional exhaled nitric oxide (FeNO) and urinary CC16;Environmental Research;2023-01
4. CC16 polymorphisms in asthma, asthma subtypes, and asthma control in adults from the Agricultural Lung Health Study;Respiratory Research;2022-11-09
5. Urinary CC16, a potential indicator of lung integrity and inflammation, increases in children after short-term exposure to PM2.5/PM10 and is driven by the CC16 38GG genotype;Environmental Research;2022-09
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