Inhibition by Lipopolysaccharide of Naloxone-Induced Luteinising Hormone Secretion Is Accompanied by Increases in Corticotropin-Releasing Factor Immunoreactivity in Hypothalamic Paraventricular Neurones in Female Rats
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Endocrine and Autonomic Systems,Endocrinology,Endocrinology, Diabetes and Metabolism
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2826.2005.01275.x/fullpdf
Reference37 articles.
1. Cytokines Act within the Brain to Inhibit Luteinizing Hormone Secretion and Ovulation in the Rat*
2. Effects of Endotoxin on in vitro Release of LHRH and Amino Acid Neurotransmitters by Preoptic Mediobasal Hypothalamic Fragments
3. Tumor Necrosis Factor-.ALPHA. Mediates Endotoxin Induced Suppression of Gonadotropin-Releasing Hormone Pulse Generator Activity in the Rat.
4. Lipopolysaccharide-Induced Suppression of the Hypothalamic Gonadotropin-Releasing Hormone Pulse Generator in Ovariectomized Goats.
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1. Acute inflammation reduces kisspeptin immunoreactivity at the arcuate nucleus and decreases responsiveness to kisspeptin independently of its anorectic effects;American Journal of Physiology-Endocrinology and Metabolism;2010-07
2. Neonatal Lipopolysaccharide Exposure Exacerbates Stress-Induced Suppression of Luteinizing Hormone Pulse Frequency in Adulthood;Endocrinology;2007-12-01
3. Endogenous opiates and behavior: 2005;Peptides;2006-12
4. Lipopolysaccharide Increases gamma-Aminobutyric Acid Synthesis in Medial Preoptic Neurones in Association with Inhibition of Steroid-Induced Luteinising Hormone Surge in Female Rats;Journal of Neuroendocrinology;2005-10
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