A family with hereditary thrombocythaemia and normal genes for thrombopoietin and c-Mpl
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2006.06316.x/fullpdf
Reference25 articles.
1. Hypersensitivity of circulating progenitor cells to megakaryocyte growth and development factor (PEG-rHu MGDF) in essential thrombocythemia
2. Translational pathophysiology: a novel molecular mechanism of human disease
3. Familial thrombocytosis in infancy presenting with a leukaemoid reaction
4. Familial essential thrombocythemia associated with a dominant-positive activating mutation of the c-MPL gene, which encodes for the receptor for thrombopoietin
5. Familial essential thrombocythemia
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1. Hereditary erythrocytosis, thrombocytosis and neutrophilia;Best Practice & Research Clinical Haematology;2014-06
2. Advances in understanding the pathogenesis of familial thrombocythaemia;British Journal of Haematology;2011-02-08
3. Association of hereditary thrombocythemia and distal limb defects with a thrombopoietin gene mutation;Blood;2009-08-20
4. Familial thrombocytosis caused by the novel germ-line mutation p.Pro106Leu in theMPLgene;British Journal of Haematology;2009-01
5. Genetic origins and clinical phenotype of familial and acquired erythrocytosis and thrombocytosis;American Journal of Hematology;2009-01
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