Stromal-derived factor-1 abolishes constitutive apoptosis of WHIM syndrome neutrophils harbouring a truncating CXCR4 mutation
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2006.06240.x/fullpdf
Reference12 articles.
1. Myelokathexis, a congenital disorder of severe neutropenia characterized by accelerated apoptosis and defective expression ofbcl-x in neutrophil precursors
2. WHIM syndromes with different genetic anomalies are accounted for by impaired CXCR4 desensitization to CXCL12
3. Small peptide inhibitors of the CXCR4 chemokine receptor (CD184) antagonize the activation, migration, and antiapoptotic responses of CXCL12 in chronic lymphocytic leukemia B cells
4. Kostmann syndrome: severe congenital neutropenia associated with defective expression of Bcl-2, constitutive mitochondrial release of cytochrome c, and excessive apoptosis of myeloid progenitor cells
5. Altered leukocyte response to CXCL12 in patients with warts hypogammaglobulinemia, infections, myelokathexis (WHIM) syndrome
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1. A phase III randomized crossover trial of plerixafor versus G-CSF for treatment of WHIM syndrome;Journal of Clinical Investigation;2023-10-02
2. Kostmann disease and other forms of severe congenital neutropenia;Acta Paediatrica;2021-06-29
3. WHIM Syndrome: from Pathogenesis Towards Personalized Medicine and Cure;Journal of Clinical Immunology;2019-07-16
4. Long-Term Outcome of WHIM Syndrome in 18 Patients: High Risk of Lung Disease and HPV-Related Malignancies;The Journal of Allergy and Clinical Immunology: In Practice;2019-05
5. How I treat warts, hypogammaglobulinemia, infections, and myelokathexis syndrome;Blood;2017-12-07
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