NKG2D-mediated immunity underlying paroxysmal nocturnal haemoglobinuria and related bone marrow failure syndromes
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2009.07795.x/fullpdf
Reference33 articles.
1. Activation of NK cells and T cells by NKG2D, a receptor for stress-inducible MICA;Bauer;Science,1999
2. Proposals for the classification of the myelodysplastic syndromes;Bennett;British Journal of Haematology,1982
3. Somatic mutations and cellular selection in paroxysmal nocturnal haemoglobinuria;Bessler;Lancet,1994
4. Impaired growth and elevated fas receptor expression in PIGA(+) stem cells in primary paroxysmal nocturnal hemoglobinuria;Chen;Journal of Clinical Investigation,2000
5. Superior growth of glycophosphatidy linositol-anchored protein-deficient progenitor cells in vitro is due to the higher apoptotic rate of progenitors with normal phenotype in vivo;Chen;Experimental Hematology,2002
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1. Case report: Immune pressure on hematopoietic stem cells can drastically expand glycosylphosphatidylinositol-deficient clones in paroxysmal nocturnal hemoglobinuria;Frontiers in Immunology;2024-01-15
2. Immune pressure on hematopoietic stem cells can drastically expand glycosylphosphatidylinositol-deficient clones in paroxysmal nocturnal hemoglobinuria;2023-09-06
3. Increased IFN‐γ+ and TNF‐α+ mucosal‐associated invariant T cells in patients with aplastic anemia;Cytometry Part B: Clinical Cytometry;2023-02-13
4. Aplastic Anemia as a Roadmap for Bone Marrow Failure: An Overview and a Clinical Workflow;International Journal of Molecular Sciences;2022-10-04
5. Does immune destruction drive all forms of bone marrow failure?;Journal of Clinical Investigation;2022-08-01
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