The production ofJAK2wild-type platelets is not downregulated in patients withJAK2V617F mutant-positive essential thrombocythaemia
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2009.07597.x/fullpdf
Reference12 articles.
1. A higher JAK2 V617F-mutated clone is observed in platelets than in granulocytes from essential thrombocythemia patients, but not in patients with polycythemia vera and primary myelofibrosis;Bellosillo;Leukemia,2007
2. The JAK2 617V>F mutation triggers erythropoietin hypersensitivity and terminal erythroid amplification in primary cells from patients with polycythemia vera;Dupont;Blood,2007
3. Long-term serial analysis of X-chromosome inactivation patterns and JAK2 V617F mutant levels in patients with essential thrombocythemia show that minor mutant-positive clones can remain stable for many years;Gale;Blood,2007
4. Platelet c-mpl expression is dysregulated in patients with essential thrombocythaemia but this is not of diagnostic value;Harrison;British Journal of Haematology,1999
5. The molecular mechanisms that control thrombopoiesis;Kaushansky;Journal of Clinical Investigation,2005
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1. Platelets from patients with myeloproliferative neoplasms have increased numbers of mitochondria that are hypersensitive to depolarization by thrombin;Scientific Reports;2023-06-06
2. Platelets from patients with myeloproliferative neoplasms have increased numbers of mitochondria that are hypersensitive to depolarization by thrombin;2023-02-21
3. Cell competition between wild-type and JAK2V617F mutant cells prevents disease relapse after stem cell transplantation in a murine model of myeloproliferative neoplasm;2021-08-26
4. Cell competition between wild-type and JAK2V617F mutant cells in a murine model of a myeloproliferative neoplasm;Experimental Hematology;2021-08
5. Cell competition between wild-type and JAK2V617F mutant cells in a murine model of a myeloproliferative neoplasm;2020-08-27
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