A molecular and cellular model to explain the differences in reactivation from latency by herpes simplex and varicella–zoster viruses
Author:
Publisher
Wiley
Subject
Physiology (medical),Neurology (clinical),Neurology,Histology,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2990.1994.tb00982.x/fullpdf
Reference51 articles.
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3. Decrease of the lymphoproliferative response to varicella-zoster virus antigen in the aged
4. Characterization of an in vivo reactivation model of herpes simplex virus from mice trigeminal ganglia
5. A herpes simplex virus type 1 latency-associated transcript mutant reactivates with normal kinetics from latent infection
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