Altered arginine metabolism in the central nervous system (CNS) of theCln3−/−mouse model of juvenile Batten disease
Author:
Publisher
Wiley
Subject
Physiology (medical),Neurology (clinical),Neurology,Histology,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2990.2008.00984.x/fullpdf
Reference89 articles.
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2. The genetic spectrum of human neuronal ceroid-lipofuscinoses;Mole;Brain Pathol,2004
3. Characterization of Cln3p, the gene product responsible for juvenile neuronal ceroid lipofuscinosis, as a lysosomal integral membrane glycoprotein;Ezaki;J Neurochem,2003
4. Biosynthesis and intracellular targeting of the CLN3 protein defective in Batten disease;Jarvela;Hum Mol Genet,1998
5. Defective intracellular transport of CLN3 is the molecular basis of Batten disease (JNCL);Jarvela;Hum Mol Genet,1999
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