In primary effusion lymphoma cells, MYB transcriptional repression is associated with v-FLIP expression during latent KSHV infection while both v-FLIP and v-GPCR become involved during the lytic cycle
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2007.06697.x/fullpdf
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1. Aberrant expression of an amplified c-myb oncogene in two cell lines from a colon carcinoma.
2. The Kaposi sarcoma–associated herpesvirus (KSHV) induces cellular interleukin 6 expression: role of the KSHV latency-associated nuclear antigen and the AP1 response element
3. Establishment and characterization of a primary effusion (body cavity- based) lymphoma cell line (BC-3) harboring kaposi's sarcoma-associated herpesvirus (KSHV/HHV-8) in the absence of Epstein-Barr virus
4. Human herpesvirus KSHV encodes a constitutively active G-protein-coupled receptor linked to cell proliferation
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1. The Role of miRNAs in Virus-Mediated Oncogenesis;International Journal of Molecular Sciences;2018-04-17
2. KSHV miRNAs Decrease Expression of Lytic Genes in Latently Infected PEL and Endothelial Cells by Targeting Host Transcription Factors;Viruses;2014-10-23
3. Combination of Arsenic and Interferon-α Inhibits Expression of KSHV Latent Transcripts and Synergistically Improves Survival of Mice with Primary Effusion Lymphomas;PLoS ONE;2013-11-08
4. Human herpesvirus 8 viral FLICE-inhibitory protein retards cell proliferation via downregulation of Id2 and Id3 expression;Molecular and Cellular Biochemistry;2010-05-29
5. KSHV/HHV8-associated lymphomas;British Journal of Haematology;2007-11-07
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