Changes in the Brain Accumulation of Glucocorticoids in abcb1a‐Deficient CF‐1 Mice-Reference-Cited by-同舟云学术

Changes in the Brain Accumulation of Glucocorticoids in abcb1a‐Deficient CF‐1 Mice

Published:2012-10-16 Issue:11 Volume:24 Page:1440-1446
ISSN:0953-8194
Container-title:Journal of Neuroendocrinology
language:en
Short-container-title:Journal of Neuroendocrinology

Author:

Mason B. L.¹²,Pariante C. M.²,Thomas S. A.¹

Affiliation:

1. Institute of Pharmaceutical Science, King’s College London, London, UK.

2. Section and Laboratory of Stress, Psychiatry and Immunology (SPI‐Lab), Institute of Psychiatry, King’s College London, London, UK.

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Endocrine and Autonomic Systems,Endocrinology,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1365-2826.2012.02353.x

Reference40 articles.

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2. AthaudaSBP AthaudaNKRB CraneA RidlonA SegalMB PrestonJE.Blood–brain barrier transfer of corticosterone in normal and protein deprived rats. Role of multi‐drug resistance protein efflux. Abstract for The Physiological Society Meeting. University of Cambridge Communications. 2004; p555.

3. Multidrug-resistance gene (P-glycoprotein) is expressed by endothelial cells at blood-brain barrier sites.

4. The three mouse multidrug resistance (mdr) genes are expressed in a tissue-specific manner in normal mouse tissues.

5. Normal viability and altered pharmacokinetics in mice lacking mdr1-type (drug-transporting) P-glycoproteins

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