Complement is not activated in ABO-haemolytic disease of the newborn
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.1988.tb04215.x/fullpdf
Reference8 articles.
1. Mechanisms of Red Cell Destruction Mediated by Non-Complement Binding IgG Antibodies: the Essential Role in Vivo of the Fc Part of IgG
2. Quantitative Measurments Concerning A and B Antigen Sites
3. Deficient Classical Complement Pathway Activity in Newborn Sera
4. C. P. Engelfriet, A. E. G. Kr. Borne, D. Beckers, F. W. Meulen, A. Fleer, D. Roos, and W. H. Ouwehand (1981 ) A Seminar on Immune-mediated Cell Destruction , chapter 5. pp.93 -130 . American Association of Blood Banks.
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1. Hemolytic Disease of the Fetus and Newborn Caused by Anti-Group A IgG From a Group B Mother;Journal of Pediatric Hematology/Oncology;2020-09-29
2. Severe ABO hemolytic disease of fetus and newborn requiring blood exchange transfusion;Asian Journal of Transfusion Science;2018
3. IMMUNOHAEMATOLOGICAL PROFILE OF MATERNAL ANTIBODY-MEDIATED ABO HAEMOLYTIC DISEASE OF FOETUS AND NEWBORN;Journal of Evolution of Medical and Dental Sciences;2017-09-07
4. ABO Hemolytic Disease of Fetus and Newborn; Still a Diagnostic Dilemma: A Case Report;Indian Journal of Hematology and Blood Transfusion;2017-02-23
5. A new preparation method for red blood cells for intrauterine transfusion enabling reduction of donor exposure;Transfusion;2015-02-05
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