Severe dominant dystrophic epidermolysis bullosa complicated by systemic amyloidosis
Author:
Publisher
Wiley
Subject
Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2133.1993.tb00278.x/fullpdf
Reference9 articles.
1. (35) Severe dominant dystrophic epidermolysis bullosa
2. Oesophageal web formation in dystrophic epidermolysis bullosa
3. Evaluation of Systemic Amyloidosis by Scintigraphy with123I-Labeled Serum Amyloid P Component
4. Metabolic studies of radioiodinated serum amyloid P component in normal subjects and patients with systemic amyloidosis.
5. Evaluation of Anchoring Fibrils and Other Components of the Dermal-Epidermal Junction in Dystrophic Epidermolysis Bullosa by a Quantitative Ultrastructural Technique
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1. Elevated expression of interleukin‐6 (IL‐6) and serum amyloid A (SAA) in the skin and the serum of recessive dystrophic epidermolysis bullosa: Skin as a possible source of IL‐6 through Toll‐like receptor ligands and SAA;Experimental Dermatology;2024-03
2. Recessive dystrophic epidermolysis bullosa (RDEB) complicated by secondary hepatic amyloidosis;JAAD Case Reports;2015-11
3. Monitoring to Prevent Complications: Anemia, Infections, Osteopenia, Failure to Thrive, Renal Disease, Squamous Cell Carcinoma, Cardiomyopathy;Blistering Diseases;2015
4. Nephro-urological complications of epidermolysis bullosa in paediatric patients;British Journal of Dermatology;2007-01
5. Recessive dystrophic epidermolysis bullosa complicated with nephrotic syndrome due to secondary amyloidosis;International Journal of Dermatology;2000-02
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