Relationships in Alzheimer's disease between the extent of Abeta deposition in cerebral blood vessel walls, as cerebral amyloid angiopathy, and the amount of cerebrovascular smooth muscle cells and collagen-Reference-Cited by-同舟云学术

Relationships in Alzheimer's disease between the extent of Abeta deposition in cerebral blood vessel walls, as cerebral amyloid angiopathy, and the amount of cerebrovascular smooth muscle cells and collagen

Published:2006-06 Issue:3 Volume:32 Page:332-340
ISSN:0305-1846
Container-title:Neuropathology and Applied Neurobiology
language:en
Short-container-title:Neuropathol Appl Neurobiol

Author:

Tian J.,Shi J.,Smallman R.,Iwatsubo T.,Mann D. M. A.

Publisher

Wiley

Subject

Physiology (medical),Clinical Neurology,Neurology,Histology,Pathology and Forensic Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2990.2006.00732.x/fullpdf

Reference36 articles.

1. beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease.

2. Amyloid ?-proteins 1?40 and 1?42(43) in the soluble fraction of extra- and intracranial blood vessels

3. Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)

4. Dominant and differential deposition of distinct β-amyloid peptide species, AβN3(pE), in senile plaques

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