Intracellular expression of type VII collagen during wound healing in severe recessive dystrophic epidermolysis bullosa and normal human skin
Author:
Publisher
Wiley
Subject
Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2133.1992.tb00447.x/fullpdf
Reference28 articles.
1. Evaluation of Anchoring Fibrils and Other Components of the Dermal-Epidermal Junction in Dystrophic Epidermolysis Bullosa by a Quantitative Ultrastructural Technique
2. Epidermolysis Bullosa Dystrophica-Recessive: A Possible Role Of Anchoring Fibrils In The Pathogenesis
3. Is There Any Specificity to Defects of Anchoring Fibrils in Epidermolysis Bullosa Dystrophica, and What Does This Mean in Terms of Pathogenesis?
4. Type VII collagen is a major structural component of anchoring fibrils.
5. Type VII Collagen is a Normal Component of Epidermal Basement Membrane, Which Shows Altered Expression in Recessive Dystrophic Epidermolysis Bullosa
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1. Epidermolysis Bullosa and Kindler Syndrome;Harper's Textbook of Pediatric Dermatology;2019-11-20
2. Identical COL71A1 heterozygous mutations resulting in different dystrophic epidermolysis bullosa phenotypes;Pediatric Dermatology;2017-12-22
3. Intraepidermal Type VII Collagen by Immunofluorescence Mapping: A Specific Finding for Bullous Dermolysis of the Newborn;Pediatric Dermatology;2017-05
4. Extracellular Matrix and Dermal Fibroblast Function in the Healing Wound;Advances in Wound Care;2016-03
5. Lysyl Hydroxylase 3 Localizes to Epidermal Basement Membrane and Is Reduced in Patients with Recessive Dystrophic Epidermolysis Bullosa;PLOS ONE;2015-09-18
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