Neuropathology of the chronic epileptic syndrome induced by intrahippocampal tetanus toxin in rat: preservation of pyramidal cells and incidence of dark cells-Reference-Cited by-同舟云学术

Neuropathology of the chronic epileptic syndrome induced by intrahippocampal tetanus toxin in rat: preservation of pyramidal cells and incidence of dark cells

Published:1992-02 Issue:1 Volume:18 Page:53-70
ISSN:0305-1846
Container-title:Neuropathology and Applied Neurobiology
language:en
Short-container-title:Neuropathol Appl Neurobiol

Author:

JEFFERYS J. G. R.,EVANS B. J.,HUGHES S. A.,WILLIAMS S. F.

Publisher

Wiley

Subject

Physiology (medical),Neurology (clinical),Neurology,Histology,Pathology and Forensic Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2990.1992.tb00764.x/fullpdf

Reference51 articles.

1. Tetanus toxin inhibits the evoked outflow of an inhibitory (GABA) and an excitatory (d-aspartate) amino acid from particulate brain cortex

2. Extracellular studies on the role of N-methyl-d-aspartate receptors in epileptiform activity recorded from the kainic acid-lesioned hippocampus

3. Temporal Lobe Volumetric Cell Densities in Temporal Lobe Epilepsy

4. Behavioural and neuropathological effects produced by tetanus toxin injected into the hippocampus of rats

5. The role of epileptic activity in hippocampal and ‘remote’ cerebral lesions induced by kainic acid

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