Normal homeostasis of fibrinolysis in nephrogenic diabetes insipidus in spite of defective V2- receptor-mediated responses of tissue plasminogen activator release
Author:
Publisher
Wiley
Subject
Clinical Biochemistry,Biochemistry,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2362.1990.tb01794.x/fullpdf
Reference29 articles.
1. Hormonal stimulation of phosphatidylinositol breakdown, with particular reference to the hepatic effect of vasopressin;Michel;Biochem Soc Trans,1979
2. Nephrogenic diabetes insipidus: close linkage with markers from the distal long arm of the human X chromosome;Knoers;Human Genet,1988
3. Linkage of X-linked nephrogenic diabetes insipidus with DXS52, a polymorphic DNA marker;Knoers;Nephron,1988
4. Specificity in the factor VIII response to vasopressin infusion in man;Prowse;Brit J Haematol,1979
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1. Agents which Increase Synthesis and Release of Tissue-Type Plasminogen Activator;Handbook of Experimental Pharmacology;2001
2. Non-Responsiveness of T-Pa, U-Pa and VWF to DDAVP;Desmopressin in Bleeding Disorders;1993
3. The Hemodynamic and Coagulant Effects of DDAVP Are Specific Extrarenal V2- Receptor Responses;Desmopressin in Bleeding Disorders;1993
4. Urokinase-type plasminogen activator release after DDAVP in von Willebrand disease: Different behaviour of plasminogen activators according to the synthesis of von Willebrand factor;Thrombosis Research;1992-06
5. Fibrinolytic response during exercise and epinephrine infusion in the same subjects;Journal of the American College of Cardiology;1992-06
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