Differential expression of stem cell markers in lichen planopilaris and alopecia areata
Author:
Publisher
Wiley
Subject
Dermatology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2133.2011.10491.x/fullpdf
Reference10 articles.
1. Histopathological evaluation of alopecias;Sellheyer;Am J Dermatopathol,2006
2. Evidence that the bulge region is a site of relative immune privilege in human hair follicles;Meyer;Br J Dermatol,2008
3. CD200, a ‘no danger’ signal for hair follicles;Rosenblum;J Dermatol Sci,2006
4. Characterization and isolation of stem cell-enriched human hair follicle bulge cells;Ohyama;J Clin Invest,2006
5. Follicular stem cell marker PHLDA1 (TDAG51) is superior to cytokeratin-20 in differentiating between trichoepithelioma and basal cell carcinoma in small biopsy specimens;Sellheyer;J Cutan Pathol,2011
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1. Destruction of the stem cell Niche, Pathogenesis and Promising Treatment Targets for Primary Scarring Alopecias;Stem Cell Reviews and Reports;2020-08-12
2. Frontal fibrosing alopecia shows robust T helper 1 and Janus kinase 3 skewing;British Journal of Dermatology;2020-06-15
3. Spiradenoma and cylindroma originate from the hair follicle bulge and not from the eccrine sweat gland: an immunohistochemical study with CD200 and other stem cell markers;Journal of Cutaneous Pathology;2014-11-24
4. Topical immunomodulation with diphenylcyclopropenone for alopecia areata: the Lebanese experience;International Journal of Dermatology;2013-10-18
5. Lichen planopilaris is characterized by immune privilege collapse of the hair follicle's epithelial stem cell niche;The Journal of Pathology;2013-09-10
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