Clinical relevance of RET variants G691S, L769L, S836S and S904S to sporadic medullary thyroid cancer
Author:
Publisher
Wiley
Subject
Endocrinology, Diabetes and Metabolism,Endocrinology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2265.2011.04293.x/fullpdf
Reference18 articles.
1. Constitutive RET tyrosine kinase activation in hereditary medullary thyroid cancer: clinical opportunities;Machens;Journal of Internal Medicine,2009
2. Medullary thyroid cancer: management guidelines of the American Thyroid Association;Kloos;Thyroid,2009
3. RET exon 11 (G691S) polymorphism is significantly more frequent in sporadic medullary thyroid carcinoma than in the general population;Elisei;Journal of Clinical Endocrinology and Metabolism,2004
4. Germline sequence variant S836S in the RET proto-oncogene is associated with low level predisposition to sporadic medullary thyroid carcinoma in the Spanish population;Ruiz;Clinical Endocrinology,2001
5. The RET polymorphic allele S836S is associated with early metastatic disease in patients with hereditary or sporadic medullary thyroid carcinoma;Siqueira;Endocrine-Related Cancer,2010
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