Role of p38 mitogen-activated protein kinase pathway on heart failure in the infant rat after burn injury
Author:
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2613.2007.00561.x/fullpdf
Reference36 articles.
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3. Tumour necrosis factor in chronic heart failure: a peripheral view on pathogenesis, clinical manifestations and therapeutic implications;Bolger;Drugs,2000
4. Cardiac molecular signaling after burn trauma;Carlson;J. Burn Care Res.,2006
5. Burn plasma mediates cardiac myocyte apoptosis via endotoxin;Carlson;Am. J. Physiol. Heart Circ. Physiol.,2002
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1. Improved heart failure by Rhein lysinate is associated with p38MAPK pathway;Experimental and Therapeutic Medicine;2018-07-09
2. Alcohol Modulation of the Postburn Hepatic Response;Journal of Burn Care & Research;2017
3. Kupffer Cell p38 Mitogen-Activated Protein Kinase Signaling Drives Postburn Hepatic Damage and Pulmonary Inflammation When Alcohol Intoxication Precedes Burn Injury;Critical Care Medicine;2016-10
4. Cardiovascular Dysfunction Following Burn Injury: What We Have Learned from Rat and Mouse Models;International Journal of Molecular Sciences;2016-01-02
5. Acceleration of skin regeneration in full-thickness burns by incorporation of bFGF-loaded alginate microspheres into a CMCS-PVA hydrogel;Journal of Tissue Engineering and Regenerative Medicine;2015-06-29
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