Induction of GITRL expression in human keratinocytes by Th2 cytokines and TNF-α: implications for atopic dermatitis-Reference-Cited by-同舟云学术

Induction of GITRL expression in human keratinocytes by Th2 cytokines and TNF-α: implications for atopic dermatitis

Published:2012-03-15 Issue:4 Volume:42 Page:550-559
ISSN:0954-7894
Container-title:Clinical & Experimental Allergy
language:en
Short-container-title:Clin Exp Allergy

Author:

Byrne A. M.¹,Goleva E.¹,Chouiali F.²,Kaplan M. H.³,Hamid Q. A.²,Leung D. Y.M.¹⁴

Affiliation:

1. Department of Pediatrics; National Jewish Health; Denver CO USA

2. Meakins-Christie Laboratories; McGill University; Montréal Québèc Canada

3. Department of Pediatrics; Herman B Wells Center for Pediatric Research; Indianapolis, IN and Department of Microbiology and Immunology; Indiana University School of Medicine; Indianapolis IN USA

4. Department of Pediatrics; University of Colorado Denver; Aurora CO USA

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2222.2012.03956.x/fullpdf

Reference39 articles.

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3. Identification of glucocorticoid-induced TNF receptor-related protein ligand on keratinocytes: ligation by GITR induces keratinocyte chemokine production and augments T-cell proliferation;Byrne;J Invest Dermatol,2009

4. Identification of a new member of the tumor necrosis factor family and its receptor, a human ortholog of mouse GITR;Gurney;Curr Biol,1999

5. Mouse glucocorticoid-induced tumor necrosis factor receptor ligand is costimulatory for T cells;Tone;Proc Natl Acad Sci USA,2003

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