Mouse homolog of SALL1, a causative gene for Townes?Brocks syndrome, binds to A/T-rich sequences in pericentric heterochromatin via its C-terminal zinc finger domains-Reference-Cited by-同舟云学术

Mouse homolog of SALL1, a causative gene for Townes?Brocks syndrome, binds to A/T-rich sequences in pericentric heterochromatin via its C-terminal zinc finger domains

Published:2007-02 Issue:2 Volume:12 Page:171-182
ISSN:1356-9597
Container-title:Genes to Cells
language:en
Short-container-title:Genes Cells

Author:

Yamashita Kazunari,Sato Akira,Asashima Makoto,Wang Pi-Chao,Nishinakamura Ryuichi

Publisher

Wiley

Subject

Cell Biology,Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2443.2007.01042.x/fullpdf

Reference46 articles.

1. Duane Radial Ray Syndrome (Okihiro Syndrome) Maps to 20q13 and Results from Mutations in SALL4, a New Member of the SAL Family

3. Detection of heterozygousSALL1 deletions by quantitative real time PCR proves the contribution of aSALL1 dosage effect in the pathogenesis of Townes-Brocks syndrome

4. Cross-regulatory interactions among tracheal genes support a co-operative model for the induction of tracheal fates in the Drosophila embryo

5. Association of Transcriptionally Silent Genes with Ikaros Complexes at Centromeric Heterochromatin

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