Kinetics of Insulin Secretion in Chronic Pancreatitis and Mild Maturity Onset Diabetes (Evidence for "Gut Hormone" Action Beyond Glucoreceptor and Cyclic Adenosine Monophosphate Mediated Insulin Release)
Author:
Publisher
Wiley
Subject
Clinical Biochemistry,Biochemistry,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2362.1976.tb00530.x/fullpdf
Reference24 articles.
1. Beta Cell Secretion—from the Standpoint of a Pathobiologist
2. Kinetics of Insulin Release from the Perfused Rat Pancreas Caused by Glucose, Glucosamine, and Galactose
3. Insulin secretion: Multifactorial regulation for a single process of release
4. The Glucose Receptor A DEFECTIVE MECHANISM IN DIABETES MELLITUS DISTINCT FROM THE BETA ADRENERGIC RECEPTOR
5. Selective Loss of Beta Cell Response to Glucose in Chronic Pancreatitis
Cited by 7 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Insulin Regulation of Hepatic Glucose Transporter Protein Is Impaired in Chronic Pancreatitis;Annals of Surgery;1994-06
2. Exocrine and Endocrine Relations in Chronic Pancreatitis;Pancreatic Enzymes in Health and Disease;1991
3. Pancreatic Structure and Glucose Tolerance in a Longitudinal Study of Experimental Pancreatitis-Induced Diabetes;Annals of Surgery;1989-08
4. Endocrine pancreatic morphology and function in exocrine insufficiency in rats;American Journal of Physiology-Gastrointestinal and Liver Physiology;1987-08-01
5. Reversal of abnormal glucose metabolism in chronic pancreatitis by administration of pancreatic polypeptide;The American Journal of Surgery;1986-01
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